- Personnellement j'ai 5 repas solides, je sais pas si c'est le cas de tout le monde ici. Dès fois je me demande si la prise avant de se coucher ne sert à rien, m'enfin on va continuer et effectivement en pratique je récupère mieux.
- Comme tu aime les études :
Ann Nutr Metab. 2006 Aug 24;50(5):476-481 [Epub ahead of print] Related Articles, Links
Actions of Short-Term Fasting on Human Skeletal Muscle Myogenic and Atrogenic Gene Expression.
Larsen AE, Tunstall RJ, Carey KA, Nicholas G, Kambadur R, Crowe TC, Cameron-Smith D.
School of Exercise and Nutrition Sciences, Deakin University, Burwood, Australia.
Background: Skeletal muscle mass is governed by multiple IGF-1-sensitive positive regulators of muscle-specific protein synthesis (myogenic regulatory factors which includes myoD, myogenin and Myf5) and negative regulators, including the atrogenic proteins myostatin, atrogin-1 and muscle ring finger 1 (MuRF-1). The coordinated control of these myogenic and atrogenic factors in human skeletal muscle following short-term fasting is currently unknown. Method: Healthy adults (n = 6, age 27.6 years) undertook a 40-hour fast. Skeletal muscle biopsy (vastus lateralis) and venous blood samples were taken 3, 15 and 40 h into the fast after an initial standard high-carbohydrate meal. Gene expression of the myogenic regulator factors (myoD, myogenin and Myf5) and the atrogenic factors (myostatin, atrogin-1 and MuRF-1) were determined by real-time PCR analysis. Plasma myostatin and IGF-1 were determined by ELISA. Results: There were no significant alterations in either the positive or negative regulators of muscle mass at either 15 or 40 h, when compared to gene expression measured 3 h after a meal. Similarly, plasma myostatin and IGF-1 were also unaltered at these times. Conclusions: Unlike previous observations in catabolic and cachexic diseased states, short-term fasting (40 h) fails to elicit marked alteration of the genes regulating both muscle-specific protein synthesis or atrophy. Greater periods of fasting may be required to initiate coordinated inhibition of myogenic and atrogenic gene expression.
Et il parle de 40 heures, mais je suppose que l'étude n'est pas sérieuse?
Je ne parle même pas de la montée de GH qui met le corps déjà en situation d'anabolisme.
De plus, comme je l'ai dis, l'étude en question a deux problèmes :
- on dort pas 12 heures.
- l'alimentation à côté n'était pas de bonne qualité, c'est indéniable et ça n'est pas rien..