par Nutrimuscle-Conseils » 27 Mar 2015 17:41
Plus que de s'inquiéter de sa protéine, il y a 2 choses à faire:
Les sels de citrate contre les calculs rénaux
Pour les personnes ayant des prédispositions à avoir des calculs rénaux, les médecins disent ce qu’il ne faut pas faire mais ne disent pas ce qu’il faut faire :
Prendre du citrate !
Impact of long-term potassium citrate therapy on urinary profiles and recurrent stone formation.
J Urol. 2009 Mar;181(3):1145-50. Robinson MR, Leitao VA, Haleblian GE, Scales CD Jr, Chandrashekar A, Pierre SA, Preminger GM.
Patients with pre-therapy and post-therapy 24-hour urinary profiles available who remained on potassium citrate for at least 6 months were included in the analysis. RESULTS: Of the 1,480 patients with 24-hour urinary profiles 503 met study inclusion criteria. Mean therapy duration was 41 months (range 6 to 168). Overall a significant and durable change in urinary metabolic profiles was noted as soon as 6 months after the onset of therapy. These changes included increased urinary pH (5.90 to 6.46, p <0.0001) and increased urinary citrate (470 to 700 mg a day, p <0.0001). The stone formation rate also significantly decreased after the initiation of potassium citrate from 1.89 to 0.46 stones per year (p <0.0001). There was a 68% remission rate and a 93% decrease in the stone formation rate. CONCLUSIONS: Potassium citrate provides a significant alkali and citraturic response during short-term and long-term therapy with the change in urinary metabolic profiles sustained as long as 14 years of treatment. Moreover, long-term potassium citrate significantly decreases the stone formation rate, confirming its usefulness in patients with recurrent nephrolithiasis.
Réduire le sodium protège des calculs rénaux
Effects of a low-salt diet on idiopathic hypercalciuria in calcium-oxalate stone formers: a 3-mo randomized controlled trial Am J Clin Nutr 91: 565-570, 2010.
Antonio Nouvenne, Tiziana Meschi, Beatrice Prati, Angela Guerra, Franca Allegri, Giuseppe Vezzoli, Laura Soldati, Giovanni Gambaro, Umberto Maggiore and Loris Borghi.
Background: A direct relation exists between sodium and calcium excretion, but randomized studies evaluating the sustained effect of a low-salt diet on idiopathic hypercalciuria, one of the main risk factors for calcium-oxalate stone formation, are still lacking. Objective: Our goal was to evaluate the effect of a low-salt diet on urinary calcium excretion in patients affected by idiopathic calcium nephrolithiasis. Design: Patients affected by idiopathic calcium stone disease and hypercalciuria (>300 mg Ca/d in men and >250 mg Ca/d in women) were randomly assigned to receive either water therapy alone (control diet) or water therapy and a low-salt diet (low-sodium diet) for 3 mo. Twenty-four-hour urine samples were obtained twice from all patients: one sample at baseline on a free diet and one sample after 3 mo of treatment. Results: A total of 210 patients were randomly assigned to receive a control diet (n = 102) or a low-sodium diet (n = 108); 13 patients (2 on the control diet, 11 on the low-sodium diet) withdrew from the trial. At the follow-up visit, patients on the low-sodium diet had lower urinary sodium (mean ± SD: 68 ± 43 mmol/d at 3 mo compared with 228 ± 57 mmol/d at baseline; P < 0.001). Concomitant with this change, they showed lower urinary calcium (271 ± 86 mg/d at 3 mo compared with 361 ± 129 mg/d on the control diet, P < 0.001) and lower oxalate excretion (28 ± 8 mg/d at 3 mo compared with 32 ± 10 mg/d on the control diet, P = 0.001). Urinary calcium was within the normal range in 61.9% of the patients on the low-salt diet and in 34.0% of those on the control diet (difference: +27.9%; 95% CI: +14.4%, +41.3%; P < 0.001). Conclusion: A low-salt diet can reduce calcium excretion in hypercalciuric stone formers.
